I have a couple questions regarding meds as being direct causes of MBP among other things. Well, I have been on antidepressants for quite some time now, and for a while I stopped taking them, around the time inbetween the ages of 16 and 17. Around the time I was almost 17 and a half I got back on them and when I turned 18 is when I started noticing a change in my hairline.
Currently im 19 and a half and the left side of my hairline has gone up maybe a bit more than half an inch and the right side about an inch. Are antidepressants directly linked to MPB? Do they trigger hair loss? Or when they talk about hair loss as a side effect are they talking about the patchy kind that just falls out and regrows when you stop taking the meds? My family history of hair loss is very inconsistant so its hard for me to tell if I really am experiencing MBP or just a mature hairline. Unfortunately I have a very skinny long face and naturally pronounced hairline so it makes it all the more noticable….. My other question is, this whole gene therapy thing. If we do find a cure for hair loss through this what will happen to your buisiness? Will you continue working for the hair loss industry? Just curious, heh. Thank you for your time sir.
While many medications may cause hair loss, the most common cause men is genes, unrelated to medication use. Genetic hair loss causes male pattern baldness (MPB). It is possible that your medications could have accelerated changes in your hairline or balding, but there is really no way to be 100% certain. Mapping out your scalp hair for miniaturization may give you insights into any gene expression that is active at this time. Male pattern baldness is also known as androgenic alopecia (AGA), because hair loss is from the effects of androgenic hormones, such as dihydrotestosterone (DHT). While we all have androgenic hormones, only the men with the gene for hair loss will lose hair. The change in your hairline, if it follows a specific pattern as MPB, will generally mean you have the gene for hair loss. To be certain though, you should be evaluated by a doctor for a physical exam and a miniaturization study. I really cannot diagnose your hair loss problem with just brief descriptions. BaldingBlog is merely a source of the opinions of our staff physicians and is not a source for diagnosis.
With respect to gene therapy, I sincerely hope it will come to fruition in my lifetime. While it is undoubtedly still many years away, it will definitely impact my field in a positive way. After all, we are the physicians in the field of hair loss. Gene therapy will only empower us to offer more options to our patients.
Steroids may accelerate the time it takes for your body to move to its final hair loss pattern. For example, if you are destined to become a Norwood Class 5A by the age of 40, the steroids may induce this pattern to develop earlier (age 25+). You will not become balder than your destined pattern. Propecia may slow down the impact of the steroids, but it will not completely stop it from occurring.
Finasteride, which is the generic name for Propecia, has been approved for the treatment of Male Pattern Hair Loss since 1998. Finasteride works by inhibiting the enzyme ,5 alpha reductase type II, that forms DHT. There has been a lot of interest by both patients and physicians in dutasteride, which is a drug that inhibits both type I and type II 5 alpha reductase enzyme.Dutasteride is three times more potent than finasteride inhibiting the type II enzyme and 100 times more potent than finasteride inhibiting the type I form of the DHT producing enzyme. Dutasteride is not approved by the FDA for the treatment of Male Pattern Hair Loss and is approved at a dose of 0.5 mg a day for the treatment of prostate enlargement. While both the type I and type II enzymes are found in the hair follicle, there is a recent study which shows that type I is present in the human brain.The function of this enzyme in the brain is still unclear.