I own a digital microscope and while there is miniaturization in the donor zone it’s not past the 20% threshold, yet at least, I would think the current pattern tends to point towards diffuse patterned alopecia. If I can bother you again, what I do want to know since information is somewhat scarce on the internet, is if there are any other causes, besides DHT, that would cause diffuse miniaturization of hair follicles? (deficiency, thyroid, allergy, mold exposure, a disease of some sort, etc.) I know most of these usually present as telogen effluvium though. Essentially, what I’m desperate to know is if miniaturization means you 100% have DHT sensitivity? I’m ready to start finasteride, but want absolute confirmation that’s what I’m dealing with before I mess with my hormones at 19. Thank you for your time.

Everyone one has less than 20% miniaturization which is normal. Each person will lose and replace up to 100 hairs per day. you have listed the many causes of diffuse thinning so check each one out with blood tests as needed, best through a doctor’s office

Thanks for the further performance pointers! Is (1) primarily avoided by trimming off as much of the skins as possible (without damaging the follicles) and (2) by inserting them so that the hairs grow out closer to perpendicular to the scalp (rather than tangential).  I speculate there is a lot of room for automation during the “filtering” the grafts based on follicle counts. I’ve read that in some areas of visual-diagnostic medicine, computer models are able to classify images more accurately than medical professionals (on a nonmedical image classification task, a convolutional neural net I built was more accurate than me in a small labeled test sample). But perhaps even more important is that it can reduce the time needed for the procedure, and therefore, also reduce the costs (which allows more people to get a transplant).

WE have monthly open hose events where our former patients come in to talk about their experience. Most people like you who come are skeptical thinking that can tell who had a transplant and they are always pleasantly surprised that they can’t tell. Good transplants can’t be detected. Look at Elan Musk before and after his transplant, a good example of a good job with actor Joe Penny (https://baldingblog.com/actor-joe-penny-shows-off-his-repaired-hairline-with-photos/).

I recently started taking finasteride(3 weeks ago) and put a post up about my fears of gynecomastia. I can confidently say thats not happening to me as my paranoia thought ( after doing some blood tests and consulting my doc). I was extremely paranoid for that too. Luckily its pretty easy to prove wrong. However, i noticed my attention started to go towards my performance with mini me (penis). I never complained about my performance for 2 weeks. Only after i stopped paying attention to my chest did this kick in. I masturbated 6 times in the last 32 hours to prove myself that i dont have ED. Each time i was able to orgasm. I noticed that it wouldn’t get fully erect when i was thinking about finasteride and its side effects. After the second go, it got more difficult to get it up ( which i expect has to do with round 1&2) I never focused on my performance prior to this so i cant really compare. So right now im not sure if i have ED or just paranoid. Essentially is there anyway i can prove that i dont have ED or do? I stopped finasteride for now until i am sure. Im aware that the percent of placebo reporting ED and finasteride ED are really close percentages. Btw if you need a more info about my performance to make a decision, please ask me. Im not shy. Morning wood is a good sign but i haven’t seen that so much since i started smoking weed b4 bed (been doing that for about a year). So what are key signs that would mean you have ED?

Jason Alexander has made a fortune with his ‘look’ and his talent. Clearly his balding was not a problem for him in the days when the TV show was popular.

It has.. from tressless

This treatment is being promoted in Germany and in Europe called AC-Therapy. It is supposed to decrease the muscle tension of the scalp which in turn decreases circulation to the scalp. Supposedly, with less circulation there is less oxygen and nutrition and this is therefore claimed as the cause for patterned hair loss in men.

We do know that the blood flow to the scalp is decreased with patterned balding, but this is because as the hair is lost or miniaturized, it needs less blood and therefore the body reduces the circulation in response to having less hair.  We also know that when a person gets a hair transplant, the blood flow increases as the demand for blood and oxygen increases to supply these new hairs with that it needs.  There is no evidence that Botox will do anything other than paralyze the scalp and forehead muscles.

The three most common causes of graft failures includes (1) exposure to air for more than 15 seconds when placing the grafts or when preparing the grafts, (2) rough handling of the grafts during placement, (3) a recipient site that is too tight.  These problems most frequently arise when the surgical team is sloppy, inexperience, poorly supervised, has no quality control systems in place implemented by the surgeon.

Pictures are worth 1000 words. Finasteride produced nice results in a relatively short time; however, I might question if the photograph is really telling the story as the results could be hair length alone.