Creatine may accelerate genetic hair loss but with conventional treatments and cessation of the creatine, it could be reversed.
In one of your posts on the blog you said you’ve done pubic hair to scalp transplants in a patient(s) before. This certainly isn’t to undermine any work done but wouldn’t a person have a bizarre appearance with pubes growing out of their head? And how well do they survive on the scalp since they are hairs with apocrine glands?
It works provided that it is not put up front and it was mixed with donor hair from the scalp. It has considerable blunk and can be hidden in the top or crown when placed appropriately. More times, however, the hair would be transplanted to the pubic area in many women who didn’t have hair there. I find it interesting that if they have hair, they shave it off, but it they don’t have public hair, they may want a transplant to have it.
Nice improvement is seen here
This is a spectacular return to your mature. dense hairline.
In your assessment of my donor supply, you noted that I can bald to a Norwood 7 and still get hair. I’m not a NW7 but out of curiosity, I know different factors are involved, like head size and stuff but how many grafts would a Norwood 7 typically require for adequate coverage at an average transplant density? I know I’ve seen photos online from another doctor in California you’ve probably heard of or maybe even know (Dr. Umar), who specializes in the use of body hair grafts. Some of the Norwood 7’s he’s restored required over 15,000 grafts! One of the clients in particular, transplanted 9,000 grafts from just his beard alone! (how many follicles are even on a man’s face because he still was able to grow a beard, albeit just much less dense); the results were pretty impressive, regardless. Granted some of those clients requested really aggressive/ juvenile hairlines and had a lot of thin/single-haired grafts harvested from their back and legs which probably accounted for the need for a higher graft harvest
I wrote an article in the Journal of Plastic and Reconstructive Surgery in 2017 which discussed that almost anyone can get a full head of hair if they combine FUE with SMP. I didn’t discuss beard hair, but the use of beard hair significantly adds to the donor supply. I believe that body hair has much less value for two reasons (1) the hair is fine, and (2) the telogen cycle is long, which results in only half of the hairs growing at any one time while the other is in telogen phase.
I was told my a dermatologist that I was balding, but nothing has changed over time as I did nothing to treat it.
You have retained most of your juvenile hairline characteristics. That is not balding, but I suspect you are slowly developing a mature hairline. This evolution, in your case, is irregular and asymmetrical, but eventually, within the next decade, possibly earlier, it will be a mature V-shaped hairline.
My question is, can miniaturized hair stabilize? What I mean by that is usually miniaturized hair continues to shrink and get smaller and narrower with each growth cycle until it eventually falls out and stop growing altogether. But can miniaturized hair miniaturize or narrow to just a certain point and then stop/stabilize at that level and not continue to the point where it eventually stops growing? I ask because I believe I have some miniaturized hairs that have been that way for over 10 years and have not changed. Also because I’ve noticed that in a lot of bald men, even those with a Norwood 7 pattern, they often aren’t “slick bald” and have some sporadic hairs remaining on top that seem to remain there permanently.Also, how do you know when hair loss as whole has stabilized on a particular person? I ask because it’s often stated/recommended for people to wait until their 30’s for a hair transplant so you have an idea of what their final pattern is and when it is stabilized. But from what I read, can’t hair loss stop, start and pick up/ slow down at different intervals throughout peoples lives?So if a person at 38 years old has a Norwood 3 pattern based on visualizing their scalp/ miniaturization studies with haircheck or whatever, and it hasn’t progressed in a few years so it’s determined for the time being it’s stable and they decide to go forward with a hair transplant; how is it known that in 2, 5, 10 years etc down the road that it won’t pic up again and other areas of the scalp won’t start miniaturizing and end up in a full Norwood 6/7 pattern or whatever?
Great questions. The concept of miniaturization leading to eventual apoptosis (death) is accepted as the standard process description. Like you, I have seen some men retain their miniaturized hair for years without growth. I expect that there is some timeline that these hairs will eventually follow the apoptosis model. With medication, the process can be slowed. I, too, have seen some Class 7 patients who have some residual both miniaturized hairs and even a few normal terminal hairs. I remember one man who gave hear of his terminal hairs a name, and when I transplanted him, he was going to check on the welfare of these named hairs. When he came back the next day, I asked him about his ‘hair’ friends, and he said: “After seeing what you gave me. I don’t care about them any longer”.
Miniaturization studies help to confirm a person’s expected balding pattern. But let’s say a person is assessed for miniaturization and their hair is miniaturized in a Norwood 3 pattern, how do you know that additional hair beyond the already miniaturized zones won’t also start to miniaturize down the road resulting in an even higher Norwood level?
How I use the donor area is a balancing act between present balding patterns, evident miniaturization suggesting future balding patterns, and the hidden future potential patterns of balding that are not evident at this time. I always keep reserves from the original donor density calculations I make on every patient.
Whatever Norwood balding pattern a person might happen to develop, does every single hair follicle in those miniaturized/ balding zones eventually miniaturize, stop growing and leave those areas (ie the hairline, crown, etc) totally hairless? Or can there be some hairs in those zones that are DHT resistant and don’t ever fall out? It seems that most balding guys I see still have some hair follicles left in the balding areas that have remained there for a while.
In the balding pattern, the hairs may all fall out (Apoptosis) or some of them may remain. I have seen Class 7 patients with a complete pattern of balding, yet when I look at the balding area, I often see a few remaining hairs here and there.
I just turned 17, and I have a family history of balding in most members of my family, both my mother’s and my father’s side. What should I do?
I assume you have genetics in the family, and you are concerned that you MIGHT follow the family pattern somehow. Just keep an eye on your hair by looking for unusual hair shedding, thinning in the front or crown, or recession of the hairline. If you should start developing these symptoms, see a good hair expert and build a complete Personalized Master Plan that you can follow over time as your hair situation changes. A good doctor will care about you, and that is important, in my opinion.
I see you mention several times in your posts that usually people who inherit the genes for MPB typically start showing signs of hair loss by 30. And for those who are destined for advanced Norwood 6/7 patterns, the pattern is usually already established/ complete by the time they are late 20’s. Those who don’t begin balding until after 30 usually develop less severe patterns like a Norwood 3. On the other hand however, I’ve seen several posts where the writer and/ or you gave examples of people who developed balding later in life and some of it to an advanced degree. What accounts for this seemingly conflicting information? Did many of those people likely already have miniaturization starting in their 20’s, but it was just never noticed and as it progressed with the age, the hairloss finally started to become evident?
Specific patients will start balding at any age making it difficult to draw conclusions on any single individual. Norwood recognized this so he looked at large populations and recorded the incidence of each pattern with respective ages. That showed most of the advanced balding patterns like the Class 7 pattern men by the age of 30. I have seen many men who started balding late in life (30s or 40s and even 50s), but those men were more exceptions to the general population statistics. If you are looking for rules to let you know where your balding pattern will go, forget the Norwood statistics and evaluate your pattern by miniaturization and your detectable hair loss
Are hair from the back of your head actually immune to dht or is this a myth?
Yes, Dr. Norman Oreintrich reported this in 1959, telling us that the band of hair reflecting the remaining hair of a Class 7 pattern is resistant to the effects of balding and DHT. That is why hair transplants work so well, as they last the lifetime of the patient.
I thought I recalled a post of yours somewhere where you said just because a person with AGA is thinning and losing hair in select areas (depending on their Norwood level) that doesn’t mean those areas will end up slick bald. Those areas can thin to a certain percentage of their original density and then stabilize there.
Thinning occurs in three forms:
1- Miniaturization which generally leads to balding. This never impacts the donor area.
2- Age-Related Thinning: generally hits older people but can hit the younger person as well. This is unified thinning all over the head, including the donor area
3- Moseac Thinning: Some people have Moseac characteristics that are almost impossible to detect without good genetic testing comparing hair follicles, a very expensive process if it can be done at all.
I was surfing the internet on topics related to hair loss and came across your balding blog which is a super helpful resource. I saw you posted on there a math formula on how to estimate the amount of grafts available in the donor area. When I looked closely with the ultra close up/ zoom option on my phone at a section of my donor and calculated the math, I average about 2.55 hairs per follicular unit which based on your formula would put me at having roughly 7,598 grafts that I can transplant. The only thing is, from what I read the diameter of you hair follicles also factors in. I don’t have a tool to measure but I’m guessing it’s more on the fine side like 45-50 microns. At 2.55 hairs per FU with the hair being more on the fine side, can you tell me how that would skew the available donor and what that would roughly bring my number to?
And, does that formula matter if it is FUE o FUT you plan to do?
The follicular unit count on the head of a typical male is 50,000 units and constant with all races and in all people. This is important to understand because the amount of hair per follicular unit is variable. Simple math would show that if two people measured 2 hairs/per follicular unit in person one and 3 hairs per follicular unit in person two, then clearly, person two would have 50% more hair to transplant. To make it a bit more confusing, if person two had a hair shaft with half of the volume of person number one, the advantage of person number two would be completely neutralized. So you see, hair numbers and hair thickness both participate in the “fullness’ we see as we look at a person’s hair volume. Dr. Marrit, in the 1980s, showed that if he plucked out half of the hairs on one side of the head in a person with black hair and medium-weight hair with an average density, the human eye could not see the difference between the two sides. This is where the surgeon’s judgment and his/her experience come in, as just a number doesn’t tell you what you must know about grafting a balding area. I can’t write a book here, but I hope this explanation helps.
Per the title. I have been taking fin for 6 months, starting at 1mg. I had sides (tiredness, lower libido) and have gradually reduced the dose to a point (0.25mg EOD) where I have no issues. Is a dose this low even worthwhile taking?
It is 50% effective as compared with the full dose. It has some value. Your call!