Dr. Rassman & associates,
What is your professional, unbiased, opinion and/or facts, as to the use of a DIM product, such as Myomin from Dr. Chi, which definitely is a known estrogen metabolizer in addition to a known aromatase inhibitor.
Dr. Wong had stated that he believes DHT is produced by excess estrogens, most notably the bad ones, as in Estradiol, in males, and further that DHT is not directly produced by testosterone but by these bad excess estrogens? Myomin and/or DIM is said to metabolize these bad estrogens and act as an aromatase inhibitor?
thanks
To put it politely, I think this Dr. Wong needs to go back and review college biochemistry if he really believes DHT is produced by excess estrogen. There are no such thing as bad estrogens, etc. DHT is NOT a byproduct of estogen or estrodiol. DHT is a byproduct of testosterone as it is metabolized. If you do not have your testicles, you will not produce enough DHT to cause balding.
It is a much more complicated process (see this chart), but here is an analogy that hopefully won’t make things too confusing:
Think of it as a one way street with a fork in the road. If you start with testosterone, you have the option of (a) going down the path of DHT or (b) going down the path of estradiol. Now it is a one way street, so you can not backtrack and go back from estradiol to testosterone to DHT. However, if there is a block in the road to estradiol, you can theoretically get a back-up in traffic and have a slight increase in testosterone, thus traffic is forced to go down the DHT pathway. Similarly, if you block the road to DHT (with finasteride) you can get a back-up and have more testosterone go down the estradiol pathway. There are other pathways, but we can ignore them for now for the sake of simplicity (if you’re not lost yet… keep up with me just a bit longer).
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