https://www.businesswire.com/news/home/20240206170882/en/Stemson-Therapeutics-Announces-Technological-Breakthrough-in-New-Hair-Growth

Hair has been cloned before. I was told that the problem encountered was when they put the cloned hairs into mice, they all died of infected hair follicles.  This was because the hairs grew in all directions and didn’t orient themselves properly; hair grew out of skin.  Think of hair as a complex organ that knows up from down and develops groups of hairs that work together in this hair organ with appropriate supportive vasculature and nerves.  If it all doesn’t go up uniformly and grows down instead (relating to the scalp surface), an infection may occur as this foreign body (the actual hair shaft is a foreign body to everyone’s scalp) causes problems.  The game isn’t over in a basketball game until the last pass is made and the clock runs out. Let’s see what they come up with.  I wish them good luck!

 

 

The miniaturization doesn’t exceed 20%, which is, therefore, by definition, normal or marginal. To be exact, repeat the photo with the hairs cut short against the scalp so that an accurate count can be made. Vary the location as well so we will be looking at more than one place.  Also map it against a photo so I know where the photos is coming from.

My situation is that I used minoxidil for 1.5 years, so imagine all that time I wasn’t a responder. Can it make my hair worse since I’m just applying alcohol all the time?  I’m asking about my situation cause I have been on fin and min and my hair is worse. 

If you used topical minoxidil, only 40% of men have the enzyme in their scalp to convert minoxidil to minoxidil sulfate, the active form of minoxidil; however, if you have taken it orally, 100% of men should get some response to the drug because the liver creates minoxidil sulfate, the active form of the drug. If you had no response to the oral, then you are not a minoxidil responder.

Hair loss has nothing to do with nutrition and this man proves it.

Weight loss doesn’t mean starvation. This man likely had achieved a good balance with fewer calories than he usually took and kept good nutrition. It is wrong to think that because this man didn’t lose his hair, others may not have been well managed during the weight loss routine. Also, this man most likely didn’t have the genes for genetic alopecia, which might have precipitated hair loss.

I can’t tell you that balding men are more likely to get divorced, but I can tell you that when divorce occurs, many men seek a hair transplant because they want to start dating and look younger.  Hair makes a man look younger. 

You know how with senile alopecia or DUPA, the hairs miniaturize but don’t (usually anyways) actually shrink to the point where they fall out and stop growing altogether? They miniaturize to a certain extent but then stabilize in a miniaturized state and are still retained (hence why you don’t see people with DUPA develop totally bald scalps because the hair is still there, just much thinner). Well, can the same thing occur with patterned hair loss? Where the hairs in the thinning areas of the scalp (vertex and/or hairline depending on the Norwood level) miniaturize to certain degree but then stabilize at that point and don’t continue to miniaturize to the point that they fall out and totally stop growing altogether? Or in patterned hair loss, miniaturizing hairs in a thinning area of the scalp will most definitely shrink to the point of no more growth?

You are drawing a lot of conclusions about DUPA. DUPA may be another form of Senile Alopecia or just a variation of genetic hair loss?  I don’t know, but I doubt it.  DUPA might progress over time, possibly leaving very little hair on the head.  i have seen this in women but never seen a man with such advanced DUPA that his entire head became very thin. DUPA is different because it impacts the Donor area and every other place on the head.  Someone can have genetically patterned hair loss and then develop DUPA, which I have seen. These men are in a tough spot, as their patterned hair loss may progress to full balding, leaving a fragile donor area. 

An Article published in the British Medical Journal in September 2016, based upon “two populations of men free of risk factors for erectile dysfunction and other sexual dysfunction or its treatment: men aged 40 or more with benign prostatic hyperplasia who received a prescription for a 5-alpha reductase inhibitor (finasteride or dutasteride) or alpha blocker, or both, and men aged 18-59 with alopecia”, suggests : “The incidence rate of erectile dysfunction was lowest among users of 5-alpha reductase inhibitors only (15.3 per 1000 person years, 95% confidence interval 14.3 to 16.5), and similar among users of 5-alpha reductase inhibitors+alpha blockers (19.2 per 1000 person years, 17.4 to 21.1) and alpha blockers only (20.1 per 1000 person years, 19.6 to 20.7). Compared with users of alpha blockers only, the adjusted incidence rate ratios for users of 5-? reductase inhibitors only and 5-alpha reductase inhibitors+alpha blockers were 0.92 (95% confidence interval 0.85 to 0.99) and 1.09 (0.99 to 1.21), respectively.” Of further note: “cases of erectile dysfunction were more likely than matched controls to be overweight or obese (as measured by body mass index) or to have a diagnosis of non-erectile dysfunction sexual dysfunction, hypertension, diabetes, hyperlipidemia, depression, orchitis, or alcohol misuse before the index date.”   Of course, this study was done in an older population that is prone to Erectile Dysfunction

https://www.bmj.com/content/354/bmj.i4823#:~:text=The%20risk%20of%20erectile%20dysfunction%20increased%20with%20increasing%20number%20of,odds%20ratios%20were%20statistically%20significant

Comment:  The risks of ED rises with age, so the groups tested were clearly at a higher risk of ED than younger men.  The statistics reported by this article suggest that in this higher-risk population, the risk of ED was 1.3%, less than was reported in the original Merck study or in the Propecia ED risk factors brochures. I am wondering why so many younger men are reporting ED, considering the above statistics?